Mast cell-mediated inflammation has been proposed as a potential contributor to neuroinflammatory insomnia and dysautonomia, but objective clinical documentation remains limited. Histamine and related immune mediators can disrupt circadian rhythm, arousal systems, and autonomic stability. Conventional pharmacological therapies for sleep restoration often fail to address these inflammatory mechanisms.
Keywords: dysautonomia, histamine, inflammation, inflammatory insomnia, mast cells, post-viral syndrome, sleep regulation
Frontiers in sleep
Case Reports
English
41640426
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