Ebola virus (EBOV) infection triggers intense host transcriptional responses that overlap extensively with those induced by other viral and bacterial pathogens. This overlap complicates the identification of EBOV-specific gene expression signatures and limits diagnostic specificity. Defining transcriptional markers that distinguish EBOV from other infections is essential for improving molecular diagnostics and advancing understanding of EBOV-specific host responses.
Keywords: Ebola virus, RNA sequencing, differential gene expression analysis, gene expression signature, host transcriptomic profiling
Frontiers in genetics
Journal Article
English
42080095
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